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This, in turn, enhances the liver’s capacity to neutralize chemical carcinogens [45,46,47], protects it from the damaging effects of various pro-oxidant xenobiotics [44,48], or enables it to compete with and outcompete-potential toxins (e.g., acrylamide) for biotransformation by GST [37].One of the mechanisms involved in antioxidant chemoprevention is the activation of the antioxidant response element (ARE), which consequently induces the expression of enzymes involved in glutathione metabolism, including not only GSTs but also GR and GPx [44,49]